Dealing for Duchenne “Rockin’ to the 80’s” Raises $120,000 for CureDuchenne 

 Annual Event Combines 80’s Fun with Fundraising to Help Find a Cure for Duchenne Muscular Dystrophy 

 San Antonio, Texas (October 30, 2024) – The San Antonio community gathered in style for the annual Dealing for Duchenne fundraiser on Saturday, October 12, 2024, at Pedrotti’s Ranch in Helotes, Texas. This year’s “Rockin’ to the 80’s” themed event raised an impressive $120,000 to support CureDuchenne’s ongoing efforts to fund research and find a cure for Duchenne muscular dystrophy, a genetic neuromuscular disease. 

The Munoz family, residents of Helotes and long-time champions for Duchenne research, once again played a vital role in the event’s success. Their son Joshua was diagnosed with Duchenne in 2013 at the age of five. Over the years, through their tireless advocacy and fundraising efforts, the Munoz family has raised over $1 million to support CureDuchenne. Their story of hope and resilience was front and center at this year’s event. 

“Watching Joshua face the challenges of Duchenne has been one of the hardest things for our family, but we’re not giving up,” said Ramiro Munoz, who co-hosted the event with his wife, Maribel. “Each year, this event gives us hope that a cure is possible, not just for Joshua, but for all children and families affected by Duchenne. We are grateful for the community’s support and to CureDuchenne for leading the way.” 

The night was filled with fun and nostalgia, as guests dressed in their best 80’s attire and enjoyed a lively casino night, dinner, drinks, and both live and silent auctions. The venue buzzed with excitement as attendees played games and bid on unique auction items, all while raising critical funds for CureDuchenne. Highlights of the evening included a special live break dance performance, 80’s musical hits and video clips, and an inspiring moment when the community came together to show their support for the Munoz family and the Duchenne community. 

Duchenne muscular dystrophy is a fatal genetic disease that affects roughly 1 in 5,000 male births. Individuals with Duchenne are typically diagnosed as toddlers, lose the ability to walk in their early-teens and often succumb to the disease in their mid-20s. Since CureDuchenne was founded in 2003, the organization has invested more than $26M in research and funded 18 research projects that have advanced to human clinical trials. In addition, CureDuchenne develops impactful programs that help families living with Duchenne around the world. 

Debra Miller, founder and CEO of CureDuchenne, expressed deep gratitude for the event’s success. “The unwavering dedication of families like the Munozes and the San Antonio community fuels our work. Every dollar raised brings us closer to treatments that can change the lives of those living with Duchenne,” Miller said. “Our mission is clear—to cure Duchenne, and with the support from events like this, we are making significant progress.” 

For more information on how you can get involved or support CureDuchenne, please visit www.cureduchenne.org. 

About CureDuchenne Twenty years ago, CureDuchenne was established with a singular mission: to find and fund a cure for Duchenne muscular dystrophy, one of the most common and severe forms of muscular dystrophy. Today, CureDuchenne is recognized as a global leader in research, patient care, and innovation, working tirelessly to improve and extend the lives of those with Duchenne. Through its innovative venture philanthropy model, CureDuchenne has advanced transformative treatments, including 18 projects that have progressed to human clinical trials. CureDuchenne’s pioneering efforts also include early funding for the first FDA-approved Duchenne drug, the creation of the first and only Duchenne-specific physical and occupational therapist certification program, and the establishment of an innovative biobank and data 

registry to accelerate research. For more information on how you can help raise awareness and funds needed for research, please visit cureduchenne.org. 

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